Vegetative Compatibility Groups of Fusarium oxysporum f. sp. lactucae from Lettuce
نویسندگان
چکیده
منابع مشابه
Local origin of two vegetative compatibility groups of Fusarium oxysporum f. sp. vasinfectum in Australia
Pathogenicity and genetic diversity of Fusarium oxysporum from geographically widespread native Gossypium populations, including a cotton growing area believed to be the center of origin of VCG 01111 and VCG 01112 of F. oxysporum f. sp. vasinfectum (Fov) in Australia, was determined using glasshouse bioassays and AFLPs. Five lineages (A-E) were identified among 856 isolates. Of these, 12% were ...
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fusarium wilt of melon is a destructive fungal disease throughout the world. in this study, the evolutionary relationships among isolates of different formae speciales of fusarium oxysporum was examined, with a special emphasis on the forma specialis melonis. bootstrapped maximum likelihood analysis of the elongation factor-1α (ef-1α) sequence was conducted on 16 iranian and 11 foreign isolates...
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Chickpea (Cicer arietinum L.) is one of the most important legume crops in Iran. Wilt disease caused by Fusarium oxysporum f. sp. ciceri, is the most important soil-borne disease of chickpea in the world. This disease caused high losses in different regions during recent years. Simple sequence repeat (SSR) were used to estimate genetic diversity in 114 of F. oxysporum isolates from six counties...
متن کاملPhylogeny of Fusarium oxysporum f. sp. lactucae Inferred from Mitochondrial Small Subunit, Elongation Factor 1-alpha, and Nuclear Ribosomal Intergenic Spacer Sequence Data.
ABSTRACT Fusarium oxysporum f. sp. lactucae, causal agent of Fusarium wilt of lettuce, is a serious pathogen recently reported in Arizona. Sequence analysis of the mitochondrial small subunit (mtSSU), translation elongation factor 1-alpha (EF-1alpha) gene, and the nuclear ribosomal DNA intergenic spacer (IGS) region was conducted to resolve relationships among f. sp. lactucae isolates, F. oxysp...
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ژورنال
عنوان ژورنال: Plant Disease
سال: 2005
ISSN: 0191-2917,1943-7692
DOI: 10.1094/pd-89-0237